Mallet finger is the term usually applied to extensor avulsion fractures. However, this entity may also be caused by distal extensor tendon ruptures. Either one results in an inability to extend the DIP joint. Mallet finger injuries are named for the resulting flexion deformity of the fingertip, which resembles a mallet or hammer. Mallet finger injuries are caused by the disruption of the extensor mechanism of the phalanx at the level of the distal interphalangeal joint usually due to a forced flexion at the distal interphalangeal joint. This injury results in the inability to extend the distal phalanx. A mallet fracture occurs when the extensor tendon also causes an avulsion of the distal phalanx. There are three subtypes of mallet fractures based on the age of the patient and the percent of the articular surface of the distal phalanx involved. The treatment remains controversial. In general, it is guided by the amount of articular surface involved in the fracture.
The distal extensor tendon is ruptured. The rupture occurs when the distal phalanx of a finger is forced into flexion while being actively extended. The extrinsic extensor tendon originates in the forearm and courses over the metacarpophalangeal joint, has an indirect attachment to the proximal phalanx, and finally, attaches to the distal phalanx. These tendons are responsible for extension of the digits. A mallet finger injury occurs when the extensor tendon is disrupted. In contrast, a mallet fracture occurs when the tendon injury causes an avulsion fracture of the distal phalanx.
Mallet finger injuries usually occur in the workplace or during sports-related activities. There is a predisposition to these injuries during participation in ball sports, as the ball hits the fingertip of an extended finger. This compels the distal interphalangeal joint into a forced flexion position and thereby causes an extensor tendon disruption. Most often, such injuries involve the long finger, ring finger or the little finger of the dominant hand. Frequently these injuries are seen in young to middle-aged men and occasionally in older women as well.
Mallet finger injuries are usually caused by a traumatic event resulting in a forced flexion of the extended fingertip. This causes a stretching or tearing of the extensor tendon. In severe injuries, this forced flexion can cause an avulsion of the tendon insertion on the distal phalanx and is described as a mallet fracture. Mallet finger injuries can also be caused by a laceration/abrasion, or more rarely, a forced hyperextension of the distal interphalangeal joint. Such an injury results in a fracture at the dorsal base of the distal phalanx. This disruption of extensor tendon function causes an unopposed flexion force on the finger and is accompanied by the inability to extend the digit. Such an injury results in the classic “mallet” appearance of the finger.
Acute mallet finger deformities are defined as those occurring within 4 weeks of the injury, while chronic mallet finger deformities occur 4 weeks after the injury.
Doyle’s Classification of Mallet Finger Injuries
The diagnosis of mallet finger injuries is usually a clinical diagnosis. Patients typically present with the history of a forced flexion injury. However, this is not always the case. Patients usually complain of pain, flexion deformity, and/or difficulty using the affected digit.
The physical examination should begin with an inspection of the soft tissue. The examination should include a full assessment of the metacarpophalangeal joint and the proximal interphalangeal joint for full range of motion. When evaluating the distal interphalangeal joint, the fingertip will rest around 45 degrees of flexion with the inability to actively extend at the distal interphalangeal joint. There may or may not be swelling and tenderness over the distal interphalangeal joint, depending on the time-frame between injury and its presentation at the emergency department.
The diagnosis of a mallet finger is usually a clinical diagnosis. However, to distinguish between mallet finger injuries and mallet fractures, physicians should obtain a three-view radiograph of the affected finger. Referring to these radiographs, physicians will either see a bony avulsion of the distal phalanx as in a mallet fracture, or the radiograph will be normal as in a mallet finger; the latter is due only to a ligamentous injury. The most valuable radiograph is the true lateral radiograph of the affected finger. This can help the physician determine the size of the fracture fragment, displacement of the fracture fragment, and subluxation of the distal phalanx.
In general, closed mallet fractures involving less than one-third of the articular surface, without associated distal interphalangeal subluxation can be managed non-surgically with splinting as the mainstay of treatment. However, the type of splint and duration of immobilization is controversial. Previously both the proximal and distal interphalangeal joints were immobilized to theoretically relax the extensor hood to promote healing of the terminal extensor tendon. However, studies on cadavers have shown that the proximal interphalangeal joint does not cause retraction of the proximal portion of the extensor tendon. Therefore, most authors advocate immobilization of only the distal interphalangeal joint. Usually, the finger is splinted in full extension (typically, 0 to 10 degrees extension) at the DIP joint for 6 to 8 weeks. This is followed by part-time splinting for an additional 4 to 6 weeks.
Numerous types of splints have been used for mallet finger injuries. These include the stack splint, the perforated thermoplastic splint, or the aluminum-foam splint. Regardless of the type of splint applied, the general principle of treatment involves full immobilization of the distal interphalangeal joint into full extension or slight hyperextension. Patients should remain in the splint continuously for 6 to 8 weeks followed by two weeks of nighttime application only. Patients should begin progressive flexion exercises following 6 weeks of immobilization. The treatment should be restarted if the distal interphalangeal joint is accidentally flexed.
Surgical management is generally accepted for mallet fractures involving greater than one-third of the articular surface and for fractures with associated joint subluxation. Many surgical techniques have also been proposed to manage mallet fractures. Similar to splinting, the general principle of surgical management of mallet fractures usually includes the placement of a Kirschner wire (K-wire) to immobilize the distal interphalangeal joint in full extension. Most surgeons suggest closed reduction and percutaneous pinning with K-wires as an alternative to open surgery to avoid complications such as pain, infection, skin irritation, and nail deformity.
The two major complications from mallet finger injuries and mallet fractures are residual extensor lag and swan neck deformities. Extensor lag is the flexion deformity that can be noted on physical exam. Swan neck deformities are due to a disruption of the volar plate caused by the disrupted extensor tendon. This results in the distal interphalangeal joint becoming abnormally flexed and the proximal interphalangeal joint remaining in a hyperextended position. Either of these complications can occur following either nonsurgical or surgical management of mallet fractures.
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