|Acute Hepatic Porphyria|
Credits: 1.00 Post-Assessment Questions: 5
Release Date: 5 Oct 2020
Expiration Date: 21 May 2021
Last Reviewed: 21 May 2020
Estimated Time To Finish: 60 Minutes
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Hepatic porphyrias are genetic disorders caused by the defect in an enzyme involved in heme synthesis and accumulation of heme precursors. Hepatic porphyrias include acute intermittent porphyria, variegate porphyria, aminolevulinic acid dehydratase deficiency porphyria, hereditary coproporphyria, and porphyria cutanea tarda. The acute hepatic porphyrias are manifest as neurological attacks (seizures, psychosis, severe abdominal and back pain, and acute polyneuropathy) and, to a lesser extent, with cutaneous manifestations such as a photosensitive blistering rash or hypertrichosis. This activity reviews the pathophysiology, evaluation, and management of the acute hepatic porphyrias and highlights the role of the interprofessional team in caring for patients affected by these conditions.
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Authors: Jiten Kothadia, Kilian LaFreniere
Editors: Jamil Shah
Editors-In-Chief: Alsadiq Al-Hillan Savio JohnHela Kchir
Chief Medical Reviewer: Abhishek Kumar
Nurse Planner/Reviewer/Editor: Lisa Haddad
Pharmacy Planner/Reviewer/Editor: Mark Pellegrini
Physician Planner/Reviewer/Editor: Scott Dulebohn
The Campbell University Jerry M. Wallace School of Osteopathic Medicine (CUSOM) is accredited by the American Osteopathic Association to provide osteopathic continuing medical education for physicians. CUSOM designates this enduring material for a maximum of 1.00 AOA Category 1 B Credits and will report CME credits commensurate with the extent of the physician's participation in the activity.
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