Mallet Finger

Article Author:
Anisha Turner
Article Editor:
Jeffrey Cooper
10/27/2018 12:31:42 PM
PubMed Link:
Mallet Finger


Mallet finger injuries are commonly encountered in everyday clinical practice. Mallet, which means hammer, was the term used to describe the hammer-like deformity that occurred in sports-related injuries in the 1800s. As some individuals do not see the hammer resemblance, some have proposed changing the name to drop the word "finger" due to its appearance. It is also known as "baseball" finger. Although it is the most common closed tendon injury seen in athletes as a result of high velocity and contact sports, it also can be the result of a relatively minor trauma such as doing household chores (tucking in a shirt, tucking in sheets) or work-related activities. Though some athletes and coaches often believe mallet injuries to be minor, each case should have a systematic evaluation performed.


The bones making up the digits are called phalanxes or phalanges. They include two bones in the thumb or three bones in the fingers as well as two to three joints between the phalanges. These joints are the distal interphalangeal joint (DIPJ); the metacarpophalangeal joint (MCPJ), which is the joint that connects the digit to the carpal or hand bones, and the proximal interphalangeal joint (PIPJ), which is the joint between the DIP and MCP joint. The joints sit in volar plates, which are collateral ligaments attached to dense fibrous connective tissue, to increase stability. Tendons are tissues that connect muscles to bone. The muscles that move the digits (fingers and thumbs) are located in the forearm and are connected to the bones of the digits by long tendons. The tendons on the top of the hand are called extensor tendons and extend or straighten the digits, while the flexor tendons on the pals side of the hand flex or bend the fingers. The tendons of the digits extend over three joints. Mallet finger injuries result when the extensor tendon is disrupted.


The most commonly affected fingers are as follows: long finger greater than ring finger greater than index finger greater than little finger, thumb. The most common cause of mallet finger is a violent flexion or laceration to the dorsum of the finger at the distal interphalangeal joint  (DIPJ).  The most common mechanism is the finger being struck at the tip or “end on” resulting in tearing of the extensor tendon where it joins at the distal phalanx, distal to the DIPJ, with or without a small fragment of bone from the insertion point.  Extensor tendon disruption causes the distal phalanx to droop or remain in a flexed position without the ability to actively extend the DIPJ, resulting in the deformity. Doyle classified mallet finger into four categories:

  • I: Closed with or without small avulsion fracture
  • II: Open laceration with tendon discontinuity
  • III: Open abrasion with skin loss
  • IV: Mallet finger
  • A: Transepiphyseal plate fracture in children
  • B: Fracture of articular surface between 20-50%
  • C: Fracture of articular surface >50%

History and Physical

Patients usually present with pain and swelling over the end of finger, the DIPJ flexed and the distal phalanx in a drooped or bent position, and inability to hold the finger straight at the DIPJ. The physical exam is pertinent for DIPJ flexion at rest, inability to straighten or extend the DIPJ during active range of motion testing, and tenderness and swelling near DIPJ. It is important to isolate the DIPJ for accurate assessment. All open injuries affecting the dorsum should raise suspicion for this type of injury as well.


Mallet finger is a clinical diagnosis that requires a thorough history and physical exam. Imaging studies are integrated as supportive measures to assess bony injuries.  An anterior-posterior (AP), lateral, and oblique view Xray centered at the DIPJ of the affected finger should be obtained to classify the mallet finger category - to differentiate a bony injury versus tendinous mallet injury. The lateral view is the most useful for assessing for avulsion fractures and volar (palmar) subluxation of the distal phalanx. Some have suggested that ultrasound may also be utilized for diagnosis.

Treatment / Management

A number of treatments have been tried, ranging from reassurance to conservative splint placements to surgically corrective procedures. Although somewhat controversial, there is some consensus in the literature that in the absence of large articular surface disruption or subluxation, non-operative treatment with placement of a splint is favorable. Yet, the type of splint, the duration of full-time wear, and the need for supplemental night orthotic wear is typically based on provider’s preference.  Options include Stax splint or an aluminum foam splint molded to create hyperextension at DIP joint. The finger should remain splinted until seen by a hand specialist.  The consensus for extension splinting duration is 6-8 weeks, with progressive flexion exercises at six weeks.  Care should be taken avoid hyperextension of the DIPJ and maintain free movement of the PIPJ. It should be encouraged to keep the splint on at all times, as removal and flexion of joint resets the 6 to 8-week clock back to time zero. Open injuries require irrigation and repair. Surgical intervention may be indicated in those who have large bone fragments, have joint mal-alignment, have Acute Type II and III mallet finger category injuries, have inadequate DIP joint extension after attempted splinting or for patients who first present several months after the initial injury. Surgical techniques include but are not limited to closed reduction with percutaneous pinning (CRPP), open reduction and internal fixation (ORIF), surgical reconstruction of the terminal tendon, and swan neck deformity correction. 

Pearls and Other Issues

Another possible complication is swan neck deformity (the DIPJ remains abnormally flexed, and the distal interphalangeal joint (PIPJ)) rests in a hyper-extended position). Swann neck deformity results from the extensor tendon disruption interfering with the volar plate’s provided stability. The disrupted extensor tendon leaves the flexor digitorum superficialis unopposed, leading to DIPJ hyperflexion while concentrated extensor on the middle phalanx results in PIPJ hyperextension. A DIPJ extensor lag of 5-10 degrees is not uncommon following treatment, as it occurs in about 40% of cases.  Approximately 70% of cases may have transient skin problems due to the splint placement.  Approximately 60% of mallet fingers have satisfactory results after splintage, followed by 20% improving in due course.


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